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A. In cancer cells, excessive production of BCL-2 sequesters and

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A. In cancer cells, excessive production of BCL-2 sequesters and
Directly targeting the mitochondrial pathway of apoptosis for cancer therapy using BH3 mimetics – recent successes, current challenges and future promise - Sarosiek - 2016 - The FEBS Journal - Wiley Online Library
A. In cancer cells, excessive production of BCL-2 sequesters and
Bcl-2-regulated cell death signalling in the prevention of autoimmunity
A. In cancer cells, excessive production of BCL-2 sequesters and
Bcl-2 family proteins and cancer
A. In cancer cells, excessive production of BCL-2 sequesters and
Targeting BCL-2-like Proteins to Kill Cancer Cells: Trends in Cancer
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Bcl-2 family members have an antiapoptotic function by blocking Bax/Bak
A. In cancer cells, excessive production of BCL-2 sequesters and
Aberrant Bcl-x splicing in cancer: from molecular mechanism to therapeutic modulation, Journal of Experimental & Clinical Cancer Research
A. In cancer cells, excessive production of BCL-2 sequesters and
Bcl-2 - an overview ScienceDirect Topics
A. In cancer cells, excessive production of BCL-2 sequesters and
Innate Conformational Dynamics Drive Binding Specificity in Anti-Apoptotic Proteins Mcl-1 and Bcl-2
A. In cancer cells, excessive production of BCL-2 sequesters and
Mechanisms of BCL-2 family proteins in mitochondrial apoptosis
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